+ |
TCL1A | up-regulates
binding
|
AKT3 |
0.485 |
Identifier |
Residue |
Sequence |
Organism |
Cell Line |
SIGNOR-81434 |
|
|
Homo sapiens |
|
pmid |
sentence |
10983986 |
Full-length tcl1 and its isoforms bind to akt / in in vitro kinase assays using gsk-3_ as a substrate, we found that the presence of any of the tcl1 family proteins (tcl1, mtcp1, or tcl1b) as gst fusion proteins significantly enhanced akt-induced gsk-3_ phosphorylation |
|
Publications: |
1 |
Organism: |
Homo Sapiens |
+ |
TCL1A | up-regulates
binding
|
AKT |
0.823 |
Identifier |
Residue |
Sequence |
Organism |
Cell Line |
SIGNOR-244449 |
|
|
Homo sapiens |
T-lymphocyte, Leukemia Cell |
pmid |
sentence |
10983986 |
Full-length tcl1 and its isoforms bind to akt / in in vitro kinase assays using gsk-3_ as a substrate, we found that the presence of any of the tcl1 family proteins (tcl1, mtcp1, or tcl1b) as gst fusion proteins significantly enhanced akt-induced gsk-3_ phosphorylation |
|
Publications: |
1 |
Organism: |
Homo Sapiens |
+ |
TCL1A | up-regulates
binding
|
AKT2 |
0.531 |
Identifier |
Residue |
Sequence |
Organism |
Cell Line |
SIGNOR-81683 |
|
|
Homo sapiens |
|
pmid |
sentence |
10983986 |
Full-length tcl1 and its isoforms bind to akt / in in vitro kinase assays using gsk-3_ as a substrate, we found that the presence of any of the tcl1 family proteins (tcl1, mtcp1, or tcl1b) as gst fusion proteins significantly enhanced akt-induced gsk-3_ phosphorylation |
|
Publications: |
1 |
Organism: |
Homo Sapiens |
+ |
TCL1A | up-regulates
binding
|
AKT1 |
0.823 |
Identifier |
Residue |
Sequence |
Organism |
Cell Line |
SIGNOR-81680 |
|
|
Homo sapiens |
T-lymphocyte, Leukemia Cell |
pmid |
sentence |
10983986 |
Full-length tcl1 and its isoforms bind to akt / in in vitro kinase assays using gsk-3_ as a substrate, we found that the presence of any of the tcl1 family proteins (tcl1, mtcp1, or tcl1b) as gst fusion proteins significantly enhanced akt-induced gsk-3_ phosphorylation |
|
Publications: |
1 |
Organism: |
Homo Sapiens |