| + |
BTF3 | up-regulates quantity by expression 
transcriptional regulation
|
HPSE2 |
0.2 |
| Identifier |
Residue |
Sequence |
Organism |
Cell Line |
| SIGNOR-253765 |
|
|
Homo sapiens |
|
| pmid |
sentence |
| 17312387 |
BTF3 silencing resulted in down-regulation of several cancer-associated genes, including EPHB2, ABL2, HPSE2 and ATM, and up-regulation of KRAG, RRAS2, NFkappa-B, MRVI1, MADCAM1 and others. |
|
| Publications: |
1 |
Organism: |
Homo Sapiens |
| + |
BTF3 | down-regulates quantity by repression 
transcriptional regulation
|
NFKB1 |
0.2 |
| Identifier |
Residue |
Sequence |
Organism |
Cell Line |
| SIGNOR-253950 |
|
|
Homo sapiens |
|
| pmid |
sentence |
| 17312387 |
In contrast, BTF3 silencing resulted in down-regulation of several cancer-associated genes, including EPHB2, ABL2, HPSE2 and ATM, and up-regulation of KRAG, RRAS2, NFkappa-B, MRVI1, MADCAM1 and others. In conclusion, BTF3 is overexpressed in PDAC, where it acts as a transcriptional regulator rather than a direct modulator of apoptosis. |
|
| Publications: |
1 |
Organism: |
Homo Sapiens |
| + |
BTF3 | down-regulates quantity by repression
transcriptional regulation
|
MADCAM1 |
0.2 |
| Identifier |
Residue |
Sequence |
Organism |
Cell Line |
| SIGNOR-253945 |
|
|
Homo sapiens |
Pancreatic Cancer Cell |
| pmid |
sentence |
| 17312387 |
In contrast, BTF3 silencing resulted in down-regulation of several cancer-associated genes, including EPHB2, ABL2, HPSE2 and ATM, and up-regulation of KRAG, RRAS2, NFkappa-B, MRVI1, MADCAM1 and others. In conclusion, BTF3 is overexpressed in PDAC, where it acts as a transcriptional regulator rather than a direct modulator of apoptosis. |
|
| Publications: |
1 |
Organism: |
Homo Sapiens |
| + |
BTF3 | up-regulates quantity by expression
transcriptional regulation
|
EPHB2 |
0.2 |
| Identifier |
Residue |
Sequence |
Organism |
Cell Line |
| SIGNOR-253949 |
|
|
Homo sapiens |
Pancreatic Cancer Cell |
| pmid |
sentence |
| 17312387 |
In contrast, BTF3 silencing resulted in down-regulation of several cancer-associated genes, including EPHB2, ABL2, HPSE2 and ATM, and up-regulation of KRAG, RRAS2, NFkappa-B, MRVI1, MADCAM1 and others. In conclusion, BTF3 is overexpressed in PDAC, where it acts as a transcriptional regulator rather than a direct modulator of apoptosis. |
|
| Publications: |
1 |
Organism: |
Homo Sapiens |
| + |
BTF3 | up-regulates quantity by expression
transcriptional regulation
|
ATM |
0.254 |
| Identifier |
Residue |
Sequence |
Organism |
Cell Line |
| SIGNOR-253948 |
|
|
Homo sapiens |
Pancreatic Cancer Cell |
| pmid |
sentence |
| 17312387 |
In contrast, BTF3 silencing resulted in down-regulation of several cancer-associated genes, including EPHB2, ABL2, HPSE2 and ATM, and up-regulation of KRAG, RRAS2, NFkappa-B, MRVI1, MADCAM1 and others. In conclusion, BTF3 is overexpressed in PDAC, where it acts as a transcriptional regulator rather than a direct modulator of apoptosis. |
|
| Publications: |
1 |
Organism: |
Homo Sapiens |
| + |
BTF3 | down-regulates quantity by repression
transcriptional regulation
|
IRAG1 |
0.2 |
| Identifier |
Residue |
Sequence |
Organism |
Cell Line |
| SIGNOR-253946 |
|
|
Homo sapiens |
Pancreatic Cancer Cell |
| pmid |
sentence |
| 17312387 |
In contrast, BTF3 silencing resulted in down-regulation of several cancer-associated genes, including EPHB2, ABL2, HPSE2 and ATM, and up-regulation of KRAG, RRAS2, NFkappa-B, MRVI1, MADCAM1 and others. In conclusion, BTF3 is overexpressed in PDAC, where it acts as a transcriptional regulator rather than a direct modulator of apoptosis. |
|
| Publications: |
1 |
Organism: |
Homo Sapiens |
| + |
BTF3 | down-regulates quantity by repression
transcriptional regulation
|
SSPN |
0.2 |
| Identifier |
Residue |
Sequence |
Organism |
Cell Line |
| SIGNOR-253951 |
|
|
Homo sapiens |
Pancreatic Cancer Cell |
| pmid |
sentence |
| 17312387 |
In contrast, BTF3 silencing resulted in down-regulation of several cancer-associated genes, including EPHB2, ABL2, HPSE2 and ATM, and up-regulation of KRAG, RRAS2, NFkappa-B, MRVI1, MADCAM1 and others. In conclusion, BTF3 is overexpressed in PDAC, where it acts as a transcriptional regulator rather than a direct modulator of apoptosis. |
|
| Publications: |
1 |
Organism: |
Homo Sapiens |
| + |
IFTAP | down-regulates activity 
binding
|
BTF3 |
0.2 |
| Identifier |
Residue |
Sequence |
Organism |
Cell Line |
| SIGNOR-260252 |
|
|
Homo sapiens |
HeLa Cell |
| pmid |
sentence |
| 18433331 |
Furthermore, we co-immunoprecipitated HEPIS with BTF3, a component of the RNA pol II initiation complex, and observed reduced proliferation of HeLa cells transfected with the HEPIS gene. |
|
| Publications: |
1 |
Organism: |
Homo Sapiens |
| + |
BTF3 | down-regulates quantity by repression
transcriptional regulation
|
RRAS2 |
0.261 |
| Identifier |
Residue |
Sequence |
Organism |
Cell Line |
| SIGNOR-253766 |
|
|
Homo sapiens |
|
| pmid |
sentence |
| 17312387 |
BTF3 silencing resulted in down-regulation of several cancer-associated genes, including EPHB2, ABL2, HPSE2 and ATM, and up-regulation of KRAG, RRAS2, NFkappa-B, MRVI1, MADCAM1 and others. |
|
| Publications: |
1 |
Organism: |
Homo Sapiens |
| + |
BTF3 | up-regulates quantity by expression
transcriptional regulation
|
ABL2 |
0.2 |
| Identifier |
Residue |
Sequence |
Organism |
Cell Line |
| SIGNOR-253947 |
|
|
Homo sapiens |
Pancreatic Cancer Cell |
| pmid |
sentence |
| 17312387 |
In contrast, BTF3 silencing resulted in down-regulation of several cancer-associated genes, including EPHB2, ABL2, HPSE2 and ATM, and up-regulation of KRAG, RRAS2, NFkappa-B, MRVI1, MADCAM1 and others. In conclusion, BTF3 is overexpressed in PDAC, where it acts as a transcriptional regulator rather than a direct modulator of apoptosis. |
|
| Publications: |
1 |
Organism: |
Homo Sapiens |
3.0
BTF3
- 
- 